- NCLEX-RN questions on tachycardias 1
- NCLEX-RN questions on tachycardias 2
- Electrical conduction in heart cells
- Normal sinus rhythm on an EKG
- Supraventricular tachycardia (SVT)
- Atrial fibrillation (Afib)
- Atrial flutter (AFL)
- Multifocal atrial tachycardia (MAT)
- Atrioventricular reentrant tachycardia (AVRT) & AV nodal reentrant tachycardia (AVNRT)
- Ventricular tachycardia (Vtach)
- Torsades de pointes
- What is ventricle fibrillation (Vfib)?
- Pulseless electrical activity (PEA) and asystole
Pulseless electrical activity (PEA) and asystole
Created by Bianca Yoo.
Want to join the conversation?
- "We never shock anyone with a pulse"...except when we do. We never defibrillate anyone with a pulse though.(5 votes)
- Is Atrial asystole also a type of cardiac arrest? I mean atrial systole is the time during which the atria contract to pump the last 30% into the ventricles.(2 votes)
- Atrial asystole would just be when the atrium is not generating any electrical impulse - this is better known as sinus node arrest but this does not generally lead to "cardiac arrest." When the atrium does not make the right electricity there are back up pulse generators in the AV junction and the ventricle. That extra "atrial kick" of blood is not necessary for life and a lot of people have problems with their atria and don't even know it.(4 votes)
- What happens when you use an AED on someone who has a non-shockable rhythm in an emergency situation?
Non-shockable rhythms don't have electrical activity so it doesn't generate a pulse but doesn't cause more harm?
I bet EMTs end up doing that.(1 vote)
- AEDs or cardiac defibrillators will not shock a patient who does not have a shockable rhythm. They are programmed to recognize Ventricular Fibrillation and Ventricular Tachycardia and will not recommend or allow a shock if the heart is not in one of these two rhythms.(4 votes)
- HOW do we differentiate between PEA and pulseless Vtach ? One is shocked and is CPR(1 vote)
- PEA will look like sinus rhythm on the ECG. PEA is not a shockable rhythm, so if you're seeing sinus rhythm but don't have a pulse you dump the shock from the defib and resume compressions.
Pulseless VT will look like VT - it'll have big, broad spikes on the ECG. VT is a shockable rhythm, so if you see VT on the monitor you deliver the shock and then resume compressions.(4 votes)
- can cancer in a different area of the heart cause a pea because of tissue mutation?(1 vote)
- There're a lot of Hs and Ts to remember. Does anyone have any tricks to remember all the Ts? As for the Hs: Very (hypoVolemia) Often (hypOxia) Puppies (low Ph, acidosis) Give (hypoGlycemia) Kindness (hypoKalemia, hyperKalemia) Therapy(hypoThermia).(1 vote)
- How would vasoconstrictors help a patient with ventricular asystole? If the heart is not beating it's not beating.(1 vote)
- Vasoconstrictors brings the blood back to heart so I am not sure if that mechanism tells the heart that it has blood it can pump around now. Besides doing vasoconstrictors, such as epinephrine and vasopressin which actually helps to conserve fluids, you give like atropine and start CPR.(1 vote)
- Is there any way to detect shockable and non shockable rythms in case of emergency when there is no ECG available at premises ? or we just do CPR in either cases? And is it of any use in case we do CPR for V Fib and V tach?(1 vote)
- Any reasonably modern defib can read a potential difference between the paddles, so they effectively have an ECG built in. CPR is essential in any non-perfusing heart rhythm - if the heart can't pump, then chest compressions are the only thing keeping the blood circulating. When you're analysing the rhythm and preparing to shock, chest compressions are the last thing to stop and the first thing to start again.(1 vote)
- treatment part is confusing .which one you treat and which you not is unclear ?(1 vote)
- How exactly does potassium concentration affect the heart cells?(1 vote)
-[Voiceover] Ventricular asystole and pulseless electrical activity are two types of cardiac arrest, meaning the heart has stopped. In both of these, a patient doesn't have a pulse, meaning that they're not pumping blood to the rest of the body, and that's why both of these conditions are absolutely fatal unless they're corrected immediately. So in ventricular asystole, there's no electrical activity in the heart. That there's no electrical activity, that means that the ventricle walls aren't contracting. And that's what asystole basically means. A means no, and systole implies ventricular contractions. So no ventricular contractions. Again, no electrical activity means no ventricular contractions, means no cardiac output, or in other words, there's no blood flowing to the rest of the body. So there's no cardiac output. And without cardiac output, you're not going to have a pulse. So anybody who has ventricular asystole will not have a pulse. On an EKG, this looks like a flat line because there's no electrical activity to cause any movement on an EKG. And this is the flat line that you hear about on movies and TV shoes, and they say, "The patient's flatlining!" It's asystole. And then there's pulseless electrical activity. And this is known as PEA. In PEA, there is electrical activity on EKG. However, it doesn't result in a pulse. And the electrical activity you see on an EKG could be something that normally produces a pulse, such as normal sinus rhythm, or even heart block, or sinus bradycardia, but for some reason there's not a pulse. Now how could this be? Well, in a normal heart, the heart's electrical activity causes the muscle cells to contract. So you have action potentials that propagate or went through the heart, and they'll lead to muscle contraction. And this relationship between electrical activity and mechanical contraction is called electro-mechanical coupling. However, when the heart is under extreme stress, so say the heart's been deprived of oxygen for a long time, the system gets disconnected. So, even though there's electrical activity, it's not going to lead to contractions. Because we've disconnected the system, and this is called electro-mechanical uncoupling. So despite the fact that cells can undergo and propagate action potentials, the action potentials don't result in muscle contraction. Another reason why you could have electrical activity without a pulse is because there could be something blocking the heart. Now the heart has a sac outlining it called the pericardium. And in some cases, this might be full of blood. And if it is full of blood, it's going to press down on the heart, and the heart is not going to have any room to pump. This is called tamponade, or cardiac tamponade. And again, this is the condition where the heart is constricted by this fluid filled sac around the heart, and the heart can't pump. So even though you can have action potentials and electrical activity in the heart, you're not going to be able to pump, and you won't have a pulse. Okay, so I'm sure you've seen in movies or TV shows, when someone flat lines or has asystole, and someone else comes running into the room, puts paddles on the chest, and yells, "Clear!" In this case, they're defibrillating the patient, meaning they're providing electrical shock to the heart, hopefully to convert the heart back to a normal rhythm. However, this is a common misconception. We never shock asystole or PEA for that matter. Defibrillation only works on very specific abnormal cardiac rhythms that can potentially be reversed with an electric shock, and these are called shockable rhythms. Shockable rhythms include ventricular fibrillation, where the walls of the ventricles are spasming, and therefore they can't contract, and they're not going to circulate blood to the rest of the body, and pulseless ventricular tachycardia, meaning that there's some sort of abnormal conduction in the ventricles that cause the ventricles to pump at a dangerously high rate. So neither of these has a pulse. And just a side note, we never shock anyone with a pulse. Something else to note is that even though ventricular fibrillation and pulseless v tach meet the criteria for PEA, and that there's electrical activity but no pulse, we don't typically classify them as PEA. These two arrhythmias are a class of their own because we treat them differently. They're shockable rhythms. PEA is considered electric activity without a pulse. That's not v fib or pulseless v tach. Now how do we treat asystole and PEA? Asystole and PEA are considered nonshockable rhythms, meaning providing a shock won't likely restore a normal rhythm. How do we treat nonshockable rhythms? We start with cardiopulmonary resuscitation, or CPR, where we alternate chest compressions with some sort of artificial breathing for the patient, whether that be mouth-to-mouth, or doing oxygen through some sort of mask. We'll also administer vasoconstrictive medications. Now, vasoconstrictive means that we're constricting the vasos, or the vessels. So these drugs constrict the blood vessels. And equally important with any patient who is pulseless, we have to consider any factors that could be reversed that might be contributing to the cardiac arrest. And you can remember which factors to consider by the mnemonic Hs and Ts, which we're going to go over in just one second. So what are the Hs and Ts? Remember, they're potentially reversible conditions that could be causing or even contributing to the cardiac arrest. So, if any patient in cardiac arrest has any of these factors, we want to try to reverse it, or fix it, to help save them. Now, something to note is that a lot of these Hs and Ts are conditions where there's some sort of lack of adequate blood circulating through the body, or adequate oxygen delivery to the body, including the heart. Heart cells need oxygen to function properly. So if there's not enough oxygen getting to the heart, then the heart cells aren't happy, and they quit working properly. The first H we're going to talk about is hypovolemia, so hypo means low, and volemia refers to volume. So this basically means low blood volume. Usually, from excessive bleeding. In hypovolemia, not enough blood is circulating, so not enough oxygen is getting around. The next H is hypoxia. That means that there's inadequate oxygen supplied to the body. And hypoxia can be due to many things, such as drowning or even a heart attack. So there's inadequate oxygen getting to the body, to the heart, and to the brain. The next H is hydrogen ions. And hydrogen ions basically means acidosis, meaning that the body's PH is too low. And acidosis often results from long periods of hypoxia. We also need to consider a person's potassium level, because hypo and hyperkalemia can lead to cardiac arrest. Now, hypokalemia means that there's too low potassium, and hyperkalemia means the potassium level is too high. Potassium plays a really important role in maintaining normal electrical conduction in the heart. So you can imagine that if the levels are too high or too low, this will disrupt the heart's electrical conduction system. In anyone with cardiac arrest that comes in, we're going to check their glucose level, because hypoglycemia, or low blood glucose, can lead to cardiac arrest, and this is something that's easily fixed. And the last H we're going to talk about is hypothermia, or low body temperature. And typically, we think of hypothermia as a temperature less than 95 degrees Fahrenheit or 35 degrees Celsius. So as a person's core temperature drops, the heart's pacemaker cells fire less and less, and eventually, the heart can stop. Okay, so those are the Hs. Now, let's move on to the Ts. We need to consider toxins, and toxins include both prescription medications and street drugs. If someone comes in because of cardiac arrest due to a toxin, there might be a reversible agent that will help reverse the effects of the toxins, and can help save the patient. The second T is tamponade, and that's something we just talked about. And we're referring to cardiac tamponade. Like we said, this is a condition where blood fills the space that lines the heart, or the pericardial sac, and this constricts the heart and makes it a lot harder for the heart to pump, and sometimes the heart can't even pump at all. And if the heart's not pumping, no blood circulating. And one way I like to think about it is to think about doing a jumping jack. Now, imagine doing that jumping jack underwater. It's a lot harder to do a jumping jack underwater because all this pressure's around your arms and legs. Likewise, it's harder for the heart to pump with all the added pressure surrounding it. And the next T is something called tension pneumothorax. So in the chest wall, the lungs are surrounded by a plural lining. So there's a space created called the plural space between the lungs and the chest wall. In a tension pneumothorax, air can somehow enter this plural space. And this is usually because of some sort of trauma to the chest. And what happens is, air enters this plural space, but it can't leave, and as more and more air enters the space, it crushes the lung, and even pushes the lung and the heart to the side. A crushed or collapsed lung is not going to be able to move oxygen very easily, and the heart can stop. And the last T we're going to talk about is thrombosis, basically meaning blood clot. In the case of cardiac arrest, we're concerned about a blood clot to the coronary artery, or an artery that supplies the heart with oxygenated blood, or we're concerned about a clot in the lungs, and that's known as a pulmonary embolism. So a pulmonary embolism is a clot in the lung. So clots in either the heart or the lungs can lead to severe oxygen depletion, and eventually lead to cardiac arrest.