- NCLEX-RN questions on tachycardias 1
- NCLEX-RN questions on tachycardias 2
- Electrical conduction in heart cells
- Normal sinus rhythm on an EKG
- Supraventricular tachycardia (SVT)
- Atrial fibrillation (Afib)
- Atrial flutter (AFL)
- Multifocal atrial tachycardia (MAT)
- Atrioventricular reentrant tachycardia (AVRT) & AV nodal reentrant tachycardia (AVNRT)
- Ventricular tachycardia (Vtach)
- Torsades de pointes
- What is ventricle fibrillation (Vfib)?
- Pulseless electrical activity (PEA) and asystole
Atrioventricular reentrant tachycardia (AVRT) & AV nodal reentrant tachycardia (AVNRT)
Created by Bianca Yoo.
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- 9:30why does the slow pathway keep stimulating the fast pathway? wouldn't it enter the refractory period after the 1st one? and mitigate this response?(4 votes)
- The slow pathway has a shorter refractory period - so it is ready by the time the signal gets through the fast pathway. The fast pathway has a longer refractory period, but the slow pathway give it enough time to recover so it can be stimulated again.(7 votes)
- What does an increase in voltage during QRS segment on EKG mean?(2 votes)
- As you probably know the QRS complex represents ventricular depolarisation. An increased voltage of QRS indicates ventricular hypertrophy (increase in volume of ventricular heart muscle). It can also be a normal finding in young people(0 votes)
- Is it correct to say that the signal is being slowed down by a built-in mecchanism at4:07?
saying it that way denotes that there is an active mechanism that slows down the signal, but it's the anatomy of this region, and it's kind of a charaterisctics of the velocity in this regione og the heart.(1 vote)
- I was teaching my Paramedic students about the pathways in the AVN and their refractory period;
The video says the alpha pathway is slow and has a short refractory period while the Mosby's textbook of paramedic(page 1144) documents that alpha pathway is slow and has a long refractory period.Kindly clarify the same.(1 vote)
- According to a study on the physiology of the dual pathways of the AV node by Mani & Pavri http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3893335/) the slow pathway has the quicker refractory period and the fast pathway has the more prolonged refractory period.(1 vote)
- what is delta wave(1 vote)
- The Greek letter "delta" is used in science & mathamatics to represent "change". In ECG, a delta wave occurs after the P-wave at the beginning of the QRS complex. It is generated by the presence of an accessory pathway called a Kent bundle and is indicative of Wolfe-Parkinson-White syndrome.
When a impulse reaches the ventricle normally it is delayed in the AV conduction system (junction) by slow decremental conduction through the compact AV node (PR interval). Once through the junction the impulse enters the His-Purkinje system where rapidly conducting tissue results in disseminated to the entire ventricle in a very short time period <120 mSec. This results in a narrow QRS complex. When a Kent bundle accessory pathway is present the impulse is delivered to the ventricle via both the normal AV conduction system where it is delayed and the Kent bundle accessory pathway which conduct rapidly to the ventricle without being delayed.
Since the accessory pathway is not delayed it begins ventricular depolarization immediately, however since it has entered the ventricle abnormally it does not use the rapidly conducting His-Purkinje system to disseminate the impulse to the tissue. It must conduct via the slow cell to cell spread of impulse through contractile tissue. This is a much slower process.
On the ECG, this absence of AV delay & slow dissemination of the impulse at the beginning of ventricular depolarization is manifested by a short or absent PR segment & a slurring of the initial portion of the QRS complex. When the same impulse makes it through the delay tissue at the AV junction it travels very rapidly to the ventricle via the His-Purkinje system so the majority of the QRS is conducted to the ventricle normally. The "change" from initial slow depolarization via the accessory pathway, to rapid conduction via the His-Purkinje system results in the slurred "Delta" or "change" wave at the beginning of the QRS. This results in:
-Short or absent PR segment
-Slurring at the onset of the QRS
-Widening of the QRS complex with the delay found at the onset(1 vote)
- In AVNRT is there a P-wave preceding the QRS complex? I tried to zoom on the video, but could not see.(1 vote)
- No, because the conduction is looping though the AV node and not depolarizing the atria. Its like how in a junctional rhythm there will often not be a P-wave. Tachycardia with a P before every QRS is sinus tachycardia and should not be treated with adenosine or cardioversion.(1 vote)
- At4:46, you state that the normal PR interval is <120msec; if your definition of the PR interval agrees with mine, which is the time between the beginning of the P wave to the beginning of the QRS complex, then a normal PR interval should be <200msec. Please let me know if I have misunderstood in any way. The PR interval isn't really necessary for understanding this topic, but thought I'd just point it out.(1 vote)
- My mom has AVNRT. Will she need a ICE?(1 vote)
- In AVRNT What significance might low Magnesiun have, to do with this also slight dehydration. ?(1 vote)
- What is the difference between micro and macro reentries?(1 vote)
Atrioventricular Re-entrant Tachycardia is also known as AVRT for short. And it's a type of supraventricular tachycardia where you have an abnormal loop of electricity, or a re-entrant circuit, going around and around between two pathways. So you have to have two pathways, one being normal AV conduction system, so that's one of the pathways. And the other pathway is an accessory pathway. And that's just an extra pathway that exists between the atrium and the ventricle. And atrioventricular re-entrant tachycardia. So I'm just going to erase this real quick, cause we're gonna re-draw that in a second. So normally, signal goes from the SA node through the atrium, to the AV node. And remember, the AV node is sort of like the gatekeeper, or the bridge that bridges signal from the atrium to the ventricle. So in a normal heart, signal has to go through this AV node. And it goes through the AV node and down to the ventricles and causes ventricular stimulation and contraction. Sometimes there's an abnormal path, or an accessory pathway, which is an extra pathway between the atrium and the ventricle. So it could be here, it could be over here, I just drew over here for convenience. But there's this extra pathway. And signal can travel from the atrium to the ventricle through this pathway and excite ventricular tissue that way. Signal can also go from the ventricles through the atrium through this accessory pathway. So it can either go forward, or in the anterograde direction, which is from the atrium to the ventricle, or backwards, which is the retrograde direction. From the ventricles to the atrium. And the direction that it goes depends on a couple of things. It depends on the timing of the refractory period of the accessory pathway. And the refractory period, remember, that's the window of time right after a group of cells are excited, in which they can't be excited again. It's kind of like a recovery period. So say you sprinted 100 meters. You're not gonna be ready to sprint another 100 meters unless you take a little break. So the refractory period is sort of like a mini-recovery period. And again, whether or not the signal goes in this forward direction or this backwards direction depends on the timing of the refractory period of the accessory pathway, as well as the direction from which signal is coming towards this accessory pathway. So if you have normal conduction through the AV node, and you have an accessory pathway, this can set you up to have a re-entrant circuit, or this abnormal loop electrical activity going around and around and around, which could cause a tachyarrhythmia. So again, you need to have both a working AV conduction system and this accessory pathway in order to have AVRT. I think one of the best ways to better conceptualize AVRT is by going through the most classic example of AVRT, which is Wolff-Parkinson-White syndrome. So again, Wolff-Parkinson-White syndrome is a classic example of AVRT. And sometimes it's called WPW for short. So again, in Wolff-Parkinson-White syndrome, you have this extra pathway or accessory pathway that exists between the atrium and the ventricle. And you have signal that goes from the SA node to the AV node, and then to the ventricles and that signal can also go from the SA node through this accessory pathway and can stimulate the ventricles that way. So you're getting ventricular stimulation through the AV node and through this accessory pathway. Now something to note is that the AV node has special tissue that actually slows down conduction so when signal hits the AV node, the conduction slows down. However, this accessory pathway is just kind of like a hole between the atrium and the ventricle. It's not gonna slow down any signal. So the signal going from the atrium to the ventricle through this pathway is actually going to stimulate ventricular cells sooner than it would the AV node, cause this AV node has this built-in mechanism that slows down conduction. So you're going to see some changes on the EKG. You're going to see a shortened PR interval, and you're gonna see the slow rise in the slope of the QRS. Again, you have this shortened PR interval, the PR interval is usually less than 0.12 seconds. And that's because you have this pre-excited ventricular tissue that's getting stimulated before the normal conduction system has a chance to stimulate ventricular tissue. And because you're getting ventricular stimulation over a longer period of time, you're gonna have this slow rise in your QRS. This slow rise is called a Delta wave. And again, this is classic for WPW. Now it's important to note that this here is not AVRT. You're not gonna get a tachyarrhythmia just from this. However, in the event that you have a premature beat coming from the SA node going to the AV node, and if this accessory pathway happens to be in a refractory period, meaning that the signal isn't gonna travel this way through the accessory pathway, then you're gonna have signal going down through the ventricles. It's gonna travel back up. And by the time it reaches the accessory pathway, it will no longer be in a refractory period. So the signal can actually travel through the accessory pathway and then go back and stimulate the AV node. This creates the re-entry circuit. So you're gonna have the signal going around and around and around and around. And this is gonna create the tachyarrhythmia that you get in AVRT. Atrioventricular Nodal Re-entrant Tachycardia is another type of re-entrant tachycardia like AVRT, but it has its differences. So people call this AVNRT for short. Remember, this is very different than AVRT. It's called AVNRT, N is for Nodal, because the abnormal loop of electricity, or that abnormal re-entrant circuit, directly involves the AV node. And the tissue right around it. There is no accessory pathway in AVNRT. So again, this is the AV node, and here I drew a bigger AV node. I kind of blew it up. So this is the AV node, and the His Bundle, and the conduction system going down into the ventricles. So in AVNRT, there are two pathways that run through the AV node. There's a slow pathway, where an impulse travels more slowly down the path, and there's a fast pathway where the impulse can zip through. Now I'm gonna erase this real quick, cause we're gonna re-draw these in a second. Okay, something else to note. Just because of this inherent make up, the slow pathway has a shorter refractory period. Now remember the refractory period is that window of time when cells can't be excited again after they've already been excited. Whereas the fast pathway has a longer refractory period. I'm gonna abbreviate refractory period RP. Refractory period. So again: slow pathway, short refractory period. Fast pathway, long refractory period. So a signal comes down, and it's gonna split. And it's gonna rush down the fast pathway, reach this common final pathway, and then spread to the ventricles. Meanwhile, it's gonna slowly go down the slow pathway. After this impulse has been transmitted through this fast pathway, it's gonna go through the refractory period. So these lines through it mean refractory period. By the time the slow pathway signal makes it to the final common pathway, it's gonna hit the refractory period of the fast pathway, and it's gonna terminate. Because no signal can be activated this way since it's in refractory period. This slow pathway is gonna enter its own refractory period, a shorter refractory period. So it's actually gonna recover. And then the fast will recover. And both the slow and fast pathway are ready for business again. They're ready for another impulse. Now let's say that there's an early beat, or a premature beat that comes in. Sometimes people call these extra beats. So there's an early beat that comes in. And let's say it comes in at a time when the fast track is still recovering from a refractory period, but the slow track has already recovered from its refractory period and is open. So this beat's gonna send impulse down the slow track. As the slow track slowly makes its way down the slow track, the fast track is gonna recover from its refractory period. So by the time the impulse reaches this final common pathway, it's gonna send signal down. And because the fast track has recovered from its refractory period, this impulse can activate the fast track and send signal back up. If the slow track has already been through its refractory period and recovered from that refractory period, it can activate the slow pathway. And send signal back down. And what happens is, the impulse will continue to circle around and around. You're creating this re-entrant loop. And as it circles around and around and around, it's gonna keep sending signal down this way. So this loop is sending signal through the AV node at a much faster rate than a normal pace maker's would, so you might see a heart rate between 100 to even 250 beats per minute. And again, it's because you have this abnormal re-entrant loop, sending signal around and around and around which is gonna spit off signal down to the ventricles at a much faster rate than a normal pacer's would. EKG is gonna look like a supraventricular tachycardia where you have a narrow QRS complex, meaning it's less than 0.12 seconds or three small boxes. And you're gonna have a heart rate of greater than or equal to 100 beats per minute. Because that's what a tachycardia is. It's greater than 100 beats per minute. So on this EKG here, you can appreciate a narrow QRS complex, and again the QRS complex is narrow because there's normal activation of the His Purkinje system, and you notice that the heart rate is greater than 100 beats per minute. So the heart rate here is somewhere between 150 and 300 beats per minute. By looking from here to here, you can tell that the heart rate is above 100 beats per minute. So this is definitely a tachycardia.